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Tirzepatide

// Compound Reference · GIP / GLP-1 Dual Agonist

Tirzepatide

CAS 2023788-19-2  ·  MW 4813.5 g/mol  ·  39 Amino Acids

A synthetic 39-amino acid peptide designed to co-activate both GIP (glucose-dependent insulinotropic polypeptide) and GLP-1 (glucagon-like peptide-1) receptors. Studied for metabolic regulation, appetite suppression, and body composition modulation in preclinical and clinical research settings.

GIP / GLP-1 Dual Agonist ≥98.5% HPLC Purity Lyophilized 5 mg / vial CoA Included Cold Shipped
Purity
≥98.5%
Molecular Weight
4813.5 g/mol
Sequence Length
39 AA
Testing Lab
Freedom Diagnostics

01 Mechanism of Action

Tirzepatide is a synthetic peptide that functions as a dual co-agonist at both GIP and GLP-1 receptors — two incretin hormone receptors expressed in the gut, pancreas, adipose tissue, and central nervous system. This dual-receptor activity distinguishes tirzepatide from single-target GLP-1 agonists such as semaglutide.

Upon receptor binding, tirzepatide activates intracellular cAMP-dependent signaling cascades that result in enhanced insulin secretion (glucose-dependent), suppressed glucagon release, delayed gastric emptying, and satiety signaling via hypothalamic pathways. GIP receptor activation additionally modulates lipid metabolism and adipogenesis at the adipocyte level — an effect absent in GLP-1-only agonists.

The molecule contains a C20 fatty diacid modification at lysine-26, enabling reversible albumin binding that extends plasma half-life to approximately five days, supporting once-weekly dosing intervals in clinical protocols.

Research Note: The additive contribution of GIP co-agonism to metabolic outcomes versus GLP-1 agonism alone remains an area of active investigation. Head-to-head clinical data indicate meaningfully greater metabolic effects from dual-agonist approaches at comparable doses.

02 Clinical Efficacy Data

The SURMOUNT-1 phase 3 trial (NCT04184622) enrolled 2,539 adults with obesity and evaluated tirzepatide at three dose levels over a 72-week treatment period. The primary endpoint was percentage change in body weight from baseline.

Dose Mean % Body Weight Change Mean Absolute Loss Responders ≥20% Loss
5 mg / week −16.0% ~35.5 lbs (16.1 kg) 30%
10 mg / week −21.4% ~48.9 lbs (22.2 kg) 50%
15 mg / week −22.5% ~52.0 lbs (23.6 kg) 57%
Placebo −2.4% ~5.3 lbs (2.4 kg) 3%

Source: SURMOUNT-1 (NCT04184622) — Jastreboff et al., NEJM 2022. Data presented for reference purposes.

Tirzepatide vs. Semaglutide — SURMOUNT-5

The SURMOUNT-5 trial (NCT05822830) conducted the first direct comparison between tirzepatide and semaglutide (2.4 mg weekly) for weight management over 72 weeks. Participants were escalated to maximum tolerated doses of each compound.

// GLP-1 Only
Semaglutide
Mean Weight Loss33.1 lbs
% Body Weight13.7%
Waist Reduction5.1 in
Relative Advantage

Source: SURMOUNT-5 (NCT05822830) — Lincoff et al., NEJM 2025.

03 Observed Research Effects

The following effects have been documented across tirzepatide clinical trials. This information is presented for research reference only and does not constitute medical guidance.

Common Reported Effects (≥5% Incidence)

Nausea
Diarrhea
Vomiting
Constipation
Abdominal discomfort
Reduced appetite
Fatigue
Dyspepsia / reflux
Injection-site reactions
Alopecia (hair thinning)

Gastrointestinal effects predominate during dose escalation and typically diminish after four to eight weeks at stable doses. Gradual titration protocols reduce GI adverse event burden in clinical settings.

Clinically Significant Signals Warranting Monitoring

  • Acute pancreatitis — Severe, persistent abdominal pain radiating to the back has been reported. Causal relationship not definitively established; pancreatitis history is a clinical consideration.
  • Gallbladder pathology — Cholelithiasis and cholecystitis observed at elevated rates in trials, consistent with rapid weight loss rather than direct drug effect.
  • Hypoglycemia risk — Elevated when co-administered with insulin secretagogues. Glucose-dependent mechanism reduces but does not eliminate risk.
  • Thyroid C-cell proliferation — Observed in rodent models at all doses; human relevance uncertain. Contraindicated in subjects with personal/family history of medullary thyroid carcinoma in clinical settings.
  • Renal function changes — Dehydration secondary to GI effects can impair renal clearance; fluid balance monitoring is indicated in sensitive populations.
  • Retinal complications — Rapid glycemic improvement in T2D subjects associated with diabetic retinopathy worsening; baseline ophthalmologic assessment recommended in diabetic research populations.
Research Use Only. The effect profile above is derived from human clinical trial data. Peplix supplies tirzepatide exclusively for in-vitro laboratory and preclinical research applications. This data is not intended to inform, guide, or support any use in living organisms.

04 Chemical & Physical Profile

Full Name Tirzepatide
CAS Number 2023788-19-2
Molecular Weight 4813.5 g/mol
Sequence Length 39 amino acids
Amino Acid Sequence Tyr-Aib-Glu-Gly-Thr-Phe-Thr-Ser-Asp-Tyr-Ser-Ile-Aib-Leu-Asp-Lys(C20-FA)-Ile-Ala-Gln-Lys-Ala-Phe-Val-Gln-Trp-Leu-Ile-Ala-Gly-Gly-Pro-Ser-Ser-Gly-Ala-Pro-Pro-Pro-Ser-NH₂
Key Modifications Aib substitutions (positions 2, 13); C20 fatty diacid at Lys-26 via γGlu-mini-PEG linker
Half-life (clinical) ~5 days (albumin-mediated)
Physical Form Lyophilized white powder
Solubility Soluble in bacteriostatic water or 0.9% saline (~1 mg/mL)
Storage −20°C (lyophilized); 2–8°C reconstituted; avoid repeated freeze-thaw
Peplix Purity ≥98.5% by HPLC
Testing Laboratory Freedom Diagnostics
Lot Number 2603
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Certificate of Analysis — Full HPLC trace and mass spectrometry data from Freedom Diagnostics is available for every Peplix batch. Contact support@peplix.bio for CoA documentation.

05 Reconstitution Reference

The following concentrations are commonly used in published tirzepatide preclinical research protocols. All calculations assume a 5 mg lyophilized vial.

Bacteriostatic Water Added Resulting Concentration Volume per 100 µg dose Doses per Vial
1.0 mL 5,000 µg/mL (5 mg/mL) 0.02 mL (20 µL) 50
2.0 mL 2,500 µg/mL (2.5 mg/mL) 0.04 mL (40 µL) 50
5.0 mL 1,000 µg/mL (1.0 mg/mL) 0.10 mL (100 µL) 50
Protocol Note: Inject bacteriostatic water slowly along the vial wall — do not inject directly onto the lyophilized cake. Swirl gently; do not vortex. Tirzepatide is moderately fragile due to its fatty acid modification. Use our Reconstitution Calculator for precise dose volumes.

06 Research Literature

The following publications form the primary evidence base for tirzepatide's pharmacology and clinical profile. All are publicly accessible via the referenced journals.

Jastreboff AM et al. Tirzepatide Once Weekly for the Treatment of Obesity. N Engl J Med. 2022;387(3):205–216. doi:10.1056/NEJMoa2206038
Lincoff AM et al. Semaglutide and Cardiovascular Outcomes in Obesity without Diabetes (SELECT). N Engl J Med. 2023. doi:10.1056/NEJMoa2307563
Frías JP et al. Tirzepatide versus Semaglutide Once Weekly in Patients with Type 2 Diabetes. N Engl J Med. 2021;385(6):503–515. doi:10.1056/NEJMoa2107519
Thomas MK et al. Dual GIP and GLP-1 Receptor Agonist — Tirzepatide. Cell Metab. 2021;33(10):1976–1987. doi:10.1016/j.cmet.2021.09.002

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≥98.5% HPLC purity · CoA from Freedom Diagnostics · Cold shipped · Lot 2603

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